by Nathan Wei, MD, FACP, FACR
Nathan Wei is a nationally known board-certified rheumatologist and author of the Second Opinion Arthritis Treatment Kit. It's available exclusively at this website... not available in stores.
Click here: Second Opinion Arthritis Treatment Kit
There is a prevailing theory among many people that arthritis is due to toxin accumulation.
A belief held by some is that environmental toxins such as food allergens and the like lead to the creation of circulating immune complexes (CIC's). This theory is controversial.
In many forms of arthritis, pain and inflammation can be caused by an overabundance of circulating immune complexes (CIC’s). Some theories purport that CIC’s are formed when molecules escape from the gastrointestinal tract due to a "leaky gut." Other antigens can be introduced as environmental pollutants that enter the body through other channels. These molecules are recognized by the immune system as foreign invaders, which lead to the formation of antibodies, which then couple with the antigen, forming the CIC.
CICs are gobbled up by cells called macrophages and then destroyed by the immune process of inflammation. However, in this process, normal or 'innocent bystander’ tissue can be damaged.
Some people support the use of antioxidants, such as Vitamin C, Vitamin E, and glutathione to mitigate the destructive effects of this inflammatory response.
Since this theory is radical, people who are proponents of this theory also advocate the use of of various herbs to detoxify organs by removing environmental toxins. Other procedures recommended include cleansing or elimination diets, fasting, colonic irrigation, saunas, lymphatic drainage techniques and homeopathic detoxification.
Herbal alteratives touted by a few include milk thistle, dandelion, gingko biloba, barberine, citrus seed, black walnut, oregano, and burdock.
One therapy that appears to be quite effective is the use of Acidophilus( Lactobacilli acidophilus). It generates lactic acid, acetic acid, and hydrogen peroxide, which can interfere with normal bowel function.
Conventional science has demonstrated that toxin exposure can be as close as one's teeth...
Relationship Between Rheumatoid Arthritis and Periodontitis
F.B. Mercado, R.I. Marshall, A.C. Klestov, and P.M. Bartold
J Periodontol 2001;72:779-787.
Background: Because of several similar features in the pathobiology of periodontitis and rheumatoid arthritis, in a previous study we proposed a possible relationship between the two diseases. Therefore, the aims of this study were to study a population of rheumatoid arthritis patients and determine the extent of their periodontal disease and correlate this with various indicators of rheumatoid arthritis.
Methods: Sixty-five consecutive patients attending a rheumatology clinic were examined for their levels of periodontitis and rheumatoid arthritis. A control group consisted of age- and gender-matched individuals without rheumatoid arthritis. Specific measures for periodontitis included probing depths, attachment loss, bleeding scores, plaque scores, and radiographic bone loss scores. Measures of rheumatoid arthritis included tender joint analysis, swollen joint analysis, pain index, physician's global assessment on a visual analogue scale, health assessment questionnaire, levels of C-reactive protein, and erythrocyte sedimentation rate. The relationship between periodontal bone loss and rheumatological findings as well as the relationship between bone loss in the rheumatoid arthritis and control groups were analyzed.
Results: No differences were noted for the plaque and bleeding indices between the control and rheumatoid arthritis groups. The rheumatoid arthritis group did, however, have more missing teeth than the control group and a higher percentage of these subjects had deeper pocketing. When the percentage of bone loss was compared with various indicators of rheumatoid arthritis disease activity, it was found that swollen joints, health assessment questionnaire scores, levels of C-reactive protein, and erythrocyte sedimentation rate were the principal parameters which could be associated with periodontal bone loss.
Conclusions: The results of this study provide further evidence of a significant association between periodontitis and rheumatoid arthritis. This association may be a reflection of a common underlying disregulation of the inflammatory response in these individuals. J Periodontol 2001;72:779-787.
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